Does lack of menstruation necessarily mean uterine adhesions?

Low menstrual flow is not necessarily caused by intrauterine adhesions, but may be related to factors such as endocrine disorders, endometrial damage, and reduced ovarian function. Intrauterine adhesions are one possible cause of light menstrual flow, but it is not the only cause.

Endocrine disorders are a common cause of scanty menstruation, mostly caused by factors such as excessive mental stress, excessive dieting, and staying up late for a long time. It manifests as menstrual cycle disorders and reduced menstrual flow. Ovarian hypofunction is more common in perimenopausal women. Due to decreased estrogen levels, endometrial hyperplasia is insufficient and menstrual flow gradually decreases. Endometrial damage is common after multiple artificial abortions and dilation and curettage. After the basal layer of the endometrium is damaged, it cannot proliferate and fall off normally. Uterine adhesions are usually secondary to uterine cavity manipulation or infection. In addition to reduced menstrual flow, they may also be accompanied by symptoms such as periodic abdominal pain and infertility.

Uterine adhesions need to be diagnosed through hysteroscopy and typically manifest as partial or complete occlusion of the uterine cavity and endometrial fibrosis. However, clinically, less menstrual flow is more related to abnormal hormone levels, such as polycystic ovary syndrome, hyperprolactinemia and other endocrine diseases. Systemic diseases such as thyroid dysfunction, anemia, and malnutrition may also cause reduced menstrual flow.

If the menstrual flow continues to decrease, you need to seek medical examination in time. The doctor will determine the cause based on the results of hormones, B-ultrasound, hysteroscopy, etc. It is necessary to maintain a regular daily routine, avoid excessive weight loss, keep the abdomen warm, and appropriately supplement iron-containing foods such as lean meat and animal liver. After intrauterine adhesions are diagnosed, they can be treated through hysteroscopic adhesion separation. After surgery, estrogen needs to be used to promote endometrial repair. If necessary, an intrauterine device should be placed to prevent re-adhesion.